Linking transcriptomic responses and phenotype in fish exposed to oil

2018-04-19 14:03:15 From: SERI Hits:

Presenter: Daniel Schlenk, Ph.D.

Venue: Meeting Room 401, Science Building 1, University Town Campus

Time: 2018-05-01, 09:30


Presenter: Professor Daniel Schlenck from The University of California Riverside;

Time: 2018-05-01, 09:30;

Venue: Meeting Room 401, Science Building 1, University Town Campus

He will give two presentations:

  1. Linking transcriptomic responses and phenotype in fish exposed to oil

  2. How to publish a scientific paper in ES&T

Daniel Schlenk, Ph.D. is Professor of Aquatic Ecotoxicology and Environmental Toxicology at the University of California Riverside. Dr. Schlenk received his PhD in Toxicology from Oregon State University in 1989. He was supported by a National Institute of Environmental Health Science postdoctoral fellowship at Duke University from 1989-1991. A Fellow of AAAS, he has served on two Scientific Advisory Panels supported by the California State Water Board in the USA focused on the monitoring of recycled and surface waters for Emerging Contaminants. Since 2016, he has been a permanent member of the USEPA Chemical Safety Advisory Committee, and from 2007-2014, he was a permanent member of the USEPA FIFRA Science Advisory Panel, which he Chaired from 2012-2014. He is currently an Associate Editor for Environmental Science and Technology, and ES&T Letters. He was co-editor-in chief of Aquatic Toxicology from 2005-2011 and currently serves on its editorial board as well as the editorial boards of Toxicological Sciences, and Marine Environmental Research.

Specialization: His research focuses on understanding the biochemical factors that influence susceptibility to environmental and natural chemicals. Four specific projects involve the impact of climate change on environmental factors that influence detoxification strategies in aquatic organisms; mechanistic investigations of the effects of salinity on pesticide toxicity in fish; the role of biotransformation as a mechanism of tolerance to natural and man-made toxic agents; impacts of endocrine disrupting chemicals on aquatic organisms; and mechanisms of PAH developmental toxicity in fish.